By M. Hegde, A. Ankala
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37, No. 1, (January 1995), pp 99-101, ISSN 03645134 Toda T, Miyake M, Kobayashi K, Mizuno K, Saito K, Osawa M, Nakamura Y, Kanazawa I, Nakagome Y, Tokunaga K & Nakahori Y. (1996) Linkage-disequilibrium mapping narrows the Fukuyama-type congenital muscular dystrophy (FCMD) candidate region to <100 kb. American Journal of Human Genetics Vol. 6 , (December 1996), pp 1313-1320, ISSN 0002-9297 Vuillaumier-Barrot S, Quijano-Roy S, Bouchet-Seraphin C, Maugenre S, Peudenier S, Van den Bergh P, Marcorelles P, Avila-Smirnow D, Chelbi M, Romero NB, Carlier RY, Estournet B, Guicheney P & Seta N.
Inclusion body formation reduces levels of mutant Huntingtin and the risk of neuronal death. Nature, Vol. 431, No. 7010,(October), pp. , Smith, MA. & Perry, G. (2003). Cerebrovascular requirement for sealant, anti-coagulant and remodeling molecules that allow for the maintenance of vascular integrity and blood supply. Brain Res Brain Res Rev, Vol. 43, No. 1, (September), pp. D. & Sawa, A. (2005). p53 mediates cellular dysfunction and behavioral abnormalities in Huntington’s disease. Neuron, Vol.
PABPN1 overexpression leads to upregulation of genes encoding nuclear proteins that are sequestered in oculopharyngeal muscular dystrophy nuclear inclusions. Neurobiol Dis, Vol. 18, No. 3 (April), pp. 551–567. , Beaudet, AL. & Zoghbi, HY. (1999). Mutation of the E6-AP ubiquitin ligase reduces nuclear inclusion frequency while accelerating polyglutamine-induced pathology in SCAI mice. Neuron, Vol. 24, No. 4, (December), pp. , Rohde, M. & Jäättelä, M. (2007). The heat shock protein 70 family: Highly homologous proteins with overlapping and distinct functions.
Muscular Dystrophy by M. Hegde, A. Ankala